AGT and lymphopenia: Not only is this a method of entry, but the virus’s interactions with the ACE2 receptor lead to an imbalance between the pro-inflammatory and anti-inflammatory effects of Angiotensin II on the Angiotensin I and Angiotensin II receptors [3]. Once inside, the virus initiates an innate and adaptive immune response which leads to several changes such as the release of several cytokines and chemokines and causes other immune changes such as lymphopenia and neutrophilia [4].