FOXJ1 and scoliosis: Taken together, these observations define a critical period in the ciliation and differentiation of normal zebrafish ChPs, identify defects in foxj1-positive ciliated cells that arise just as katnb1mh102/mh102 mutants develop scoliosis, and suggest that abnormal ChP development may contribute to CSF flow defects and spinal curve formation in katnb1mh102/mh102 mutants.