Infection with WT GBS resulted in significantly enhanced production of GM-CSF, IL-7, IL-17, IP-10, LIF, LIX, MCP-1, and RANTES in the uterus (Supplemental Fig. 6) and eotaxin, GM-CSF, IL-1α, IL-15, IL-17, LIF, LIX, MCP-1, M-CSF, and MIG in the decidua (Supplemental Fig. 7) compared to uninfected controls (*P < 0.05, one-way ANOVA, #P < 0.05, Student’s t test). The gene discussed is IL1A; the disease is infection.