Our results indicate that infection with WT GBS results in significantly enhanced production of IL-1β, KC, and TNF-α in the uterus, decidua, placenta, amnion, and fetus (Supplemental Fig. 5) compared to uninfected controls (*P < 0.05, one-way ANOVA, #P < 0.05, Student’s t test). This evidence concerns the gene IL1B and infection.