There are reports suggesting relatively frequent presence of NSAbs in patients with slowly progressing cognitive impairment or movement disorders mimicking neurodegenerative diseases, for example, Alzheimer disease (LGI1),41, 42 frontotemporal dementia (voltage‐gated potassium channels, VGKC‐Abs),43 amyotrophic lateral sclerosis (GABAB),44 and several NSAbs were found in patients diagnosed with Creutzfeldt–Jakob disease.45 This evidence concerns the gene LGI1 and amyotrophic lateral sclerosis.