As expected, genes associated with AML proliferation (FLT3, KIT), leukemia stem cells (CD34, CD38, and IL1RAP), and candidate genes overexpressed in AML (CD99, CD200, and LAMP2) were downregulated after chemotherapy, consistent with recent scRNA-Seq and immunohistochemistry data (31). Here, CD38 is linked to acute myeloid leukemia.