Using an antibody recognizing phospho-Tau S385 in mice, corresponding to S396 in human, we observed lower levels of phosphorylated Tau in hearts lacking ErbB2 and/or ERRα, suggesting that this proteotoxic stress is not an underlying cause of the observed DCM in these mice (Supplementary Fig. 7b). This evidence concerns the gene MAPT and familial dilated cardiomyopathy.