The elevation of oncomarkers may raise suspicion of malignancy being implicated: conversely, CEA levels are heightened in many forms of lung injury such as in idiopathic pulmonary fibrosis and in active smokers [45]; moreover, no cases of adenocarcinoma were reported by the authors at extended follow-up in patients with elevated CEA who survived. The gene discussed is CEACAM5; the disease is pulmonary fibrosis.