The experimental data showed that CCmDHNPs suppressed and facilitated apoptosis by modulating protein expression of B-cell lymphoma-2 (Bcl-2) and poly (ADP-ribose) polymerase (PARP), producing sound inhibitory effects on xenograft tumors in nude mice, as well as effectively suppressed invasion, metastasis, and adhesion of breast cancer cells by regulating the levels of E-cadherin, N-cadherin, MMP2, CD44, and Snail molecules. The gene discussed is CD44; the disease is breast carcinoma.