ALDHhigh CSC levels were found to be higher in TNBC tissues and cell lines than in tissue obtained from other breast cancer subtypes.[30] Many studies have found that CD44 and PD‐L1 exhibit higher expression levels in CSCs than in differentiated TNBC;[24] thus, PmTriTNE might better target CSCs, and we have shown that TriTNE could efficiently eliminate CSCs in vitro. The gene discussed is CD274; the disease is breast carcinoma.