ALDH1A2 and atherosclerosis: The key findings of other articles included (1) macrophage Aldh1a2, a gene involved in the metabolism of retinoic acid, was depressed with anti-miR-33 treatment, resulting in the activation of Treg and protection against atherosclerosis (67); and (2) netrin-1 as a neuronal guidance cue that mediates chemorepulsion and chemoreattraction of axons through receptor UNC5b, promotes atherosclerotic plaque progression through the retention of macrophages within inflamed blood vessels (68).