SLC11A2 and myocardial infarction: huc-MSC-exos deliver miR-23a-3p to cardiomyocytes in mice with acute myocardial infarction (AMI) and inhibit the expression of divalent metal transporter 1 (DMT1), thereby increasing GSH levels and decreasing the production of ROS and malondialdehyde (MDA); however, no significant effects on the levels of GPX4 were reported [48].