Consistent with the histological findings, OA could not restore the serum levels of in Scr, BUN, the expressions of kidney injury markers (Kim-1 and Ngal), apoptosis-related genes (Bax and Bcl-2), OXPHO-related genes (Atp5a1 and Ndufa) and FAO-related genes (ACSL-1 and ACADM), or BNIP3 in AKI mice (Figures 7C–J). Here, LCN2 is linked to acute kidney injury.