NFKB1 and arthropathy: These observations demonstrated that miR-150-3p could effectively regulate the Trim14/NF-κB/IFN-β axis of the innate immune response and stimulate FLSs to influence chondrocytes and the co-culture microenvironment, indicating that miR-150-3p may be a potential therapeutic target in OA and other joint diseases (Figure 9).