Genes whose expression was restored to control levels with pargyline treatment in comparison to the diabetic condition (clusters 8 and 10) are involved in the modulation of ubiquitin-mediated proteolysis, glycolysis and gluconeogenesis, oxidative stress, fluid shear stress and atherosclerosis, calcium cycling in cardiac cells, protein processing within the endoplasmic reticulum, phagocytosis, cell–cell interaction, mammalian target of rapamycin (mTOR) signaling, regulation of cytoskeleton and regulation of cardiomyocyte hypertrophy through miRNAs. Here, MTOR is linked to atherosclerosis.