Diabetes is a disease that triggers chronic hyper-inflammatory conditions [1,21,22,23], impairing the activity and presence of neutrophils [1], monocytes and dendritic cells [24] that result in pro-inflammatory and chemotactic mediators’, such as IL-6, MCP-1 (CCL2), IL-1, TNF-α, IL-4 and IFN-γ, persistent secretion [25,26,27,28,29]. Here, CCL2 is linked to diabetes mellitus.