MYD88 and peritonitis: A disruption of the intestinal barrier upon injury to intestinal epithelial cells is known to lead to the exposure of numerous TLR ligands produced by commensals and eventually may induce intestinal inflammation but also peritonitis, and mice lacking TLR2, TLR4, or the myeloid differentiation primary response 88 (MyD88) are more sensitive to these diseases [32,33,34,35].