Based on gene ontologies and differential GE, the analysis revealed seven tumour clusters, with six stable clusters each displaying a distinctive gene signature, namely: the mesenchymal (M), mesenchymal stem-like (MSL), basal like-1 (BL-1) and -2, (BL-2), luminal androgen receptor (LAR), immunomodulatory (IM) subtype, and one additional unstable (UNS) tumour cluster that expresses genes that are found across the other six clusters [14]. This evidence concerns the gene PTPRF and neoplasm.