Lastly, in CR-TNBC patient P942, the baseline PDX tumor (BTY14) had high phosphorylation on the shared pTyr sites of SRC, FYN, LCK, YES1, and FGR as well as SFK substrates such as tensin, SHIP-1 (INPP5D), AFAP1L2, paxillin, and PTK2. This evidence concerns the gene AFAP1L2 and neoplasm.