In addition, KEGG pathway analysis revealed that the upregulated DEPs mostly participated in the PPAR signaling pathway, chemical carcinogenesis, retinol metabolism, glycine, serine, and threonine metabolism, fatty acid elongation, biosynthesis of unsaturated fatty acids, glutathione metabolism, steroid hormone biosynthesis, hepatocellular carcinoma, arachidonic acid metabolism, and drug metabolism-cytochrome P450 (Figure 4f). This evidence concerns the gene PPARA and hepatocellular carcinoma.