In this study, phenotypic analysis of G6PD enzyme activity indicated that 3.6% (18/498) of the study participants had < 30% enzyme activity of the AMM, which is stated as G6PD deficiency; and 56 female patients had intermediate G6PD enzyme activity (30–80%) of AMM with heterozygous gene mutation. The gene discussed is G6PD; the disease is G6PD deficiency.