Recently, AI-Salam et al. [17] have identified GAL3 as an anti-apoptotic mediator in cardiomyocytes at 24 h post-myocardial infarction, and they further demonstrated that high GAL3 after myocardial infarction has a protective role on the heart through modulating lysosomal Cathepsins in ischaemic myocardium [18]. Here, LGALS3 is linked to myocardial infarction.