Of note, a recent study explored the hypothesis that pre‐natal maternal immune dysfunction associated with increased childhood asthma risk (revealed by low IFN‐γ:IL‐13 secretion during the third trimester of pregnancy) alters immune training at birth through epigenetic mechanisms and promotes early‐life airway colonization by asthmagenic microbiota.44 The gene discussed is IFNG; the disease is asthma.