MAOB and Parkinson disease: Beyond Parkinson’s disease, a trend for steady decline of astrogliosis is supported by 11C-DED PET data (targeting the MAO-B) in carriers of autosomal dominant Alzheimer’s disease.46 Within a preserved region, it is possible, that astrocytes (in Parkinson’s disease as well as autosomal dominant Alzheimer’s disease), partly, retain the mechanism of beneficial α-synuclein clearance—reference to comparable 11C-BU99008 VT values in HC groups.