It is noteworthy that CDK2 was found to be significantly upregulated in a panel of ATLL-derived and HTLV-1-transformed cell lines compared to activated T-cell controls (72); low miR-150 levels might thus contribute to sustain growth of HTLV-1-infected cells by relaxing control of CDK2 expression. The gene discussed is CDK2; the disease is adult T-cell leukemia/lymphoma.