For instance, AURKA overexpression through reduced proteolysis has been observed in head and neck [12] and breast [13] cancer cells, and a new study reports that undegraded AURKA at mitotic exit enhances fragmentation of the mitochondrial network in the following interphase [14], with fragmented mitochondrial networks being a characteristic of some cancer cells, including human invasive breast cancer [15]. This evidence concerns the gene AURKA and invasive breast carcinoma.