To gain a more comprehensive understanding of the regulatory mechanisms of BAF60c in AAA, we measured the expression of contractile gene Myh11, inflammatory genes Ccl2 and Il6, and the antiapoptotic gene Bcl2 in the suprarenal abdominal aortas from Baf60cfl/flApoe–/– and Baf60cSMKOApoe–/– mice after saline or Ang II (1,000 ng/kg/min) infusion for 7 days. The gene discussed is BCL2; the disease is triple-A syndrome.