Compared to the uninjured mice, we found that the mRNA expression levels of Gria3, Gria4, Grin2b, and Grin2c were markedly increased in DCN at day 7 post-SNC in non-ataxia control and ataxia mice (Supplementary Fig. 10a, b). The gene discussed is GRIN2C; the disease is cerebellar ataxia.