SLPI reduced the intracellular ROS level in ischemia/reperfusion injury in cardiomyocyte and endothelial cell experiments (13, 14), while products of oxidative stress including protein carbonyl (PC) and ischemia-modified albumin (IMA) were used as oxidative stress markers in patients with myocardial ischemia after percutaneous coronary intervention and left ventricular hypertrophy (39, 40). This evidence concerns the gene ALB and myocardial ischemia.