Nevertheless, the subtype-specific structural disruption of each of the three same-cell coexistent mSWI/SNF variants by silencing of known subtype-specific and essential structural subunits [16] showed a clear proliferative dependency of neuroblastoma cells on BAF complex structural integrity, which was observed when silencing the pair of redundant and mutually exclusive BAF-specific subunits ARID1A/B (Fig. 1D, Fig. S1A-B), and also after ARID1B silencing in the ARID1A-defficient COG-N-278 neuroblastoma cell line (Fig. S1C-D). The gene discussed is ARID1B; the disease is neuroblastoma.