Isolation of all complex subtypes existing in neuroblastoma cell lines by immunoprecipitation of the ubiquitous BRG1 catalytic subunit and analysis of its interactors by mass spectrometry led to the identification of all known mSWI/SNF subunits from the core and ATPase modules, as well as those specific to the three same-cell coexistent variants (Fig. 1A, Table S1), except from BRM (or SMARCA2), which is mutually exclusive with BRG1, the bait of the proteomic study. The gene discussed is SMARCA2; the disease is neuroblastoma.