Bmal1 deficiency in the human carotid aggravated intracellular ROS accumulation and endothelial‐to‐mesenchymal transition through the bone morphogenetic protein‐mediated signaling, demonstrating the central role of Bmal1 loss in atherosclerosis progression (Zhu et al., 2018). The gene discussed is BMAL1; the disease is atherosclerosis.