VCAM1 and cancer: EMT is highly relevant for TEM [36] and the mechanism by which NLGN1 induces TEM could rely on the activation of an EMT program, by which cancer cells adhere to the endothelium and cross the vessel wall, perhaps by forming pseudopodia and invadopodia, as it is the case , for example, for Vascular cell adhesion molecule 1 (VCAM1) [36].