We previously showed that CD163, that is the scavenger receptor for the hemoglobin‐haptoglobin complex, is upregulated in BMDMs infiltrating GBM tissues,11 and thus we evaluated its expression on GBM cell suspensions at protein level and observed a high proportion of BMDMs co‐expressing CD163 and HO‐1, as opposed to MG cells (Figure 1A), thus reinforcing the notion that BMDMs have a pronounced iron metabolism in the TME of GBM. Here, CD163 is linked to glioblastoma.