NFKB1 and diabetic kidney disease: Accumulating evidence indicates that consistent low grade of sterile inflammation is pivotal in the pathophysiology of DKD, including proinflammatory cytokines excretion, the chemokines and adhesive molecules recruitment, as well as nuclear factor κB (NF-κB) activation (Alicic et al., 2017; Rayego-Mateos et al., 2020; Liu et al., 2021).