These results agree with those obtained inbreast cancer, where Bcl2L10 has been reported to promote its anti-apoptoticfunction by negatively regulating the release of calcium from the endoplasmicreticulum through the interaction of the Bcl-2 homology (BH)4 domain withinositol-1,4,5-trisphosphate receptor [12]. This evidence concerns the gene BCL2L10 and cancer.