Of note, by using human melanoma cells stablyoverexpressing Bcl2L10 or transiently transfected for loss/gainfunction of Bcl2L10, we evidenced that Bcl2L10 is an inducer of invitro cell invasion/migration and VM, similarly to what was observedfor other anti-apoptotic proteins, such as Bcl-2 and Bcl-xL [4, 31]. Here, BCL2L1 is linked to melanoma.