TRPV1 and cardiac hypertrophy: Chronic dietary capsaicin intake can increase the proficiency of mitochondrial complex I OXPHOS, production of ATP, expression of cardiac mitochondrial sirtuin 3, and activity of complex I enzyme activity via activating TRPV1, leading to alleviation in cardiac mitochondrial dysfunction and prevention of cardiac hypertrophy induced by a high-salt diet (Lang et al., 2015).