SARS-CoV-2-related neurodegenerations, especially nigrostriatal degenerations, have been proposed owing to the possibility of neuro-invasion via the olfactory bulb, Angiotensin-Converting Enzyme 2 (ACE2) receptor expression by dopaminergic neurons, associated inflammatory and vascular changes that overlap with PD pathogenesis, and progression in addition to reported cases of SARS-CoV-2-related parkinsonism with dopamine transporter imaging abnormalities1,24. The gene discussed is SLC6A3; the disease is Parkinsonism.