Similar to our previous results, CCL18, CCL26, and LIGHT had the most dramatic effects in promoting OXTRLow CAFs-induced HN6 invasion (Fig. 5j), but only knockdown of CCL26, and with a lesser extent of CCL18, impaired OXTRHigh CAFs-dependent tumor invasion (Fig. 5k). The gene discussed is TNFSF14; the disease is neoplasm.