We believe this is because most PM lesions, regardless of tumor size, have an hypoxic environment due to high intratumor pressure caused by abundant fibrotic stroma, which obstructs blood perfusion.23 In terms of the adipocyte environment, there was no difference in CD36 expression between the lesions on the adipocyte-rich visceral peritoneum, such as greater omentum or mesentery, and those on the adipocyte-poor parietal peritoneum. The gene discussed is CD36; the disease is neoplasm.