TP53 and juvenile Huntington disease: The KEGG pathway analysis indicated enrichment in the ribosome, cell cycle, oxidative phosphorylation, DNA replication, oocyte meiosis, proteasome, pyrimidine metabolism, base excision repair, Huntington disease, Parkinson disease, homologous recombination, p53 signaling cascade, nonalcoholic fatty liver disease, progesterone-mediated oocyte maturation, spliceosome, mismatch repair, Fanconi anemia cascade, nucleotide excision repair, etc. (Figure 4H).