Similarly, several studies showed that B4galnt2 can modify the sialylated surface receptors for avian influenza strains, thereby masking the attachment site for these viruses and inhibiting infection (Heaton et al., 2017; Wong et al., 2019), further emphasizing an important function of B4galnt2-glycosylation for infection with bacterial and viral pathogens. This evidence concerns the gene B4GALNT2 and infection.