In one report, for instance, Cao et al. [37] determined that miR-150 was able to suppress SRCIN1 translation in lung cancer, thereby functioning in an oncogenic manner, while Sharma et al. [38] revealed that SRCIN1 was capable to suppress tumor cell growth and invasion via suppressing Src or E-cadherin/EGFR signaling. This evidence concerns the gene EGFR and lung cancer.