Chen et al. (19) found that the interaction of LINCOO152 with EZH2 inhibits interleukin-24 (IL24) transcription to promote lung adenocarcinoma proliferation, and ectopic expression of IL24 partially reversed the LAD cell growth promotion induced by LINC00152 overexpression (19)(Figure 4). This evidence concerns the gene CYTOR and lung adenocarcinoma.