After erastin- and sulfasalazine-induced ferroptosis of AML cells, the mRNA and protein expression of SLC7A11 increased, as revealed by the qRT-PCR and Western blot results; this finding is similar to that of previous studies on other tumors (35–37), suggesting that erastin and sulfasalazine may induce adaptive cellular responses in AML cells under stress conditions. The gene discussed is SLC7A11; the disease is acute myeloid leukemia.