Although STAT5 has not been included in Figure 2, as it is not yet confirmed to be a potent oncogene in solid tumors, an increased expression level of STAT5 was notably found in lung cancer cells, promoting the survival of cancerous cells via the tyrosine-protein kinase ABL2 and transcriptional coactivator TAZ signaling axis (26, 27). This evidence concerns the gene STAT5A and lung cancer.