We previously reported that commensal bacteria shaped immune surveillance efficiency in B16/F10 melanoma and Lewis lung carcinoma, while another group provided evidence that the microbiota promoted lung cancer development induced by the Kras mutation and the loss of p53; interestingly, both effects involved the regulation of the microbiota on γδT17 cells [4, 17]. The gene discussed is KRAS; the disease is Carcinoma, Lewis Lung.