AHR affected pro-inflammatory cytokines IL-1β, IL-6, IFN-γ, CXCL1, as well as S100β, NGN2, and NGN1 gene and protein expression after MCAO. TMF treatment modulated gene and protein expression related to neurogenesis after stroke, leading an increased proliferation of neural progenitor cells at the ipsilesional neurogenic zones. The gene discussed is IL1B; the disease is stroke disorder.