EFR3A and classic Hodgkin lymphoma: Additionally, milk yield, body condition, and subclinical ketosis have all been linked to CHL development (Amory et al., 2008; Green et al., 2014; Sepúlveda-Varas et al., 2018), so KCNQ3 and EFR3A may also contribute to the genetic correlation between DCT and sole lesions.