It was found that m6A abundantly existed in ITGA6 transcripts, and methyltransferase-like 3 (METTL3) promoted YTHDF1/YTHDF3 binding to the ITGA6 mRNA 3′-UTR, thereby enhancing ITGA6 mRNA translation and changing the adhesion ability of bladder cancer cells (Laudato et al., 2017; Jin H. et al., 2019). The gene discussed is METTL3; the disease is urinary bladder cancer.