The same peptide did not elicit any immune response in PBMC cells from healthy HLA-A*24:02+ donors (Mizukoshi et al., 2011), leading the authors to suggest a mechanism in which the potentially naturally occurring glycosylation of the serine residue at peptide position 6 could be omitted in cancer cells (Bennun et al., 2013; Peixoto et al., 2019), resulting in the formation of a unglycosylated neoepitope. This evidence concerns the gene HLA-A and cancer.