CD274 and acute respiratory distress syndrome: Anti-PD-L1 antibody was administered intraperitoneally into the murine model of ARDS and showed that lung injury, inflammatory factors, BALF protein concentration, BALF total cell count and neutrophil count were significantly lower in the anti-PD-L1 antibody group compared with those in the isotype antibody group (Figures 7A–E), indicating that treatment with anti-PD-L1 antibody attenuated lung injury and inhibited inflammatory factors, protein exudation and neutrophil infiltration of lung tissue.